Saturday, May 4, 2013

TMD and the EAR and EYE Connection


TMD and the EAR and EYE Connection




To fully appreciate the TMD and the EAR and EYE connection, it would benefit to look at their commonality in nerve innervations.



The Trigeminal Nerve (V): It is further subdivided into three branches:

1.      Ophthalmic Division (V1): It is the upper division of the Trigeminal nerve system. It has three major branches:
a.       Lacrimal nerve cries sensory information from the lateral part of the upper eyelid, conjunctiva, and lacrimal gland.
b.      Frontal
c.       Nasociliary
2.      Maxillary Division (V2): It is the middle division of the Trigeminal nerve and gives general sensory to the nerve branches to the zygomatic, infraorbital areas (branching further off to the outer nasal, upper lip areas), upper teeth, eye, palatal, posterior nasal areas, pharyngeal area and the meningies.
3.      Mandibular Division (V3): It is the lower division of the Trigeminal nerve and gives sensory the buccal, tongue, lower teeth, frontal ear and acoustic areas, the temporomandibular joint areas and meningies.
a.       It also caries motor branches to medial pterygoid (further to Tensor Veli Palatini and Tensor Tympani), lateral pterygoid, masseter, deep temporal, front area of the digastric and mylohyoid muscles.

The Facial Nerve (VII): Innervates muscles of the nose, buccinators, rhisorius, obicularis oris, muscles of lower lip and chin, platysma, and posterior area of the digastric.

There are three types of Pain:
1.      Vascular------- 10%
2.      Neurologic---- 10%
3.      Muscular------ 90%

The primary etiology of muscle pain is due to sustained muscle contraction resulting in vascular entrapment. Blood flow is impaired, oxygen is depleted and anaerobic metabolism starts.

The EAR Connection:


Eustachian Tube dysfunction: The Eustachian tube is the connection between the middle ear and the pharynx. It has three functions which are the aeration (equalizing pressure), clearance, and protection of the middle ear. Pressure equalization being its main option is regulated by three muscles including the Tensor Veli Palatini (TVP).
Patients suffering form TMJD often complain of a hearing loss, fullness isn the ears, and hypersensitivity to sound (hyperacusis). The TVP is innervated by the V3 division of the trigeminal nerve (motor branch) and lies in close anatomical proximity to the medial pterygoid. When there is pain occurring from primary pain source such as the temporalis, masseters, and medial pterygoid muscles, the effects spread to other muscles that share the same nerve supply. Dysfunction in muscles causes excitation and resulting in unnecessary sustained contraction of a muscle. In the case of the TVP muscle this sustained contraction causes the lumen of the Eustachian tube to remain patent at rest and pressure build up. This will give rise to the complaint of fullness and pressure in the ear and hollow sounding voice, but no changes in hearing. The closeness of the TVP to the medial Pt muscle is of great importance. Any chronic contraction of the medial Pt can impair normal function of the TVP. This in turn will prevent normal dilation of the Eustachian tube resulting in an inability to equalize the middle and outer ear pressure. Patients may complain of ear pain in a rapidly descending airplane and when scuba diving.

Displaced Condyle (retruded) and Mandibular-Malleolar Ligament (MML): A change in vertical dimension affects condylar position within the glenoid fossa. Teeth dictate the relationship of the lower jaw (mandible) to the upper jaw (maxilla) when teeth are in contact. A deep bite/over closure will translate the condyles back within the glenoid fossa resulting in compression of the retrodiscal tissue. The patient’s impression is pain in the ear (otalgia). The MML also plays a role in ear complaints. This ligament connects the neck and anterior process of the Malleous (hammer shaped bone in the middle ear, part of the ear ossicles) to a part of the capsule, the disc, and sphenomandibular ligament (ligament between the sphenoid bone and the mandible) through the small channel known as the petrotympanic fissure. A change in mandibular posture can affect the tension of the MML which in turn affects the movement of the ear ossicles. Many patients will complain of pressure and/or ringing in the ears when moving the lower jaw forward. The petrotympanic fissure is a communication from the middle ear to the glenoid fossa (the cavity where the joint is) and it is locate behind the condyle. In addition the fissure also contains the anterior tympanic artery which supplies the tympanic membrane (ear drum) and chorda tympani (branch of the Facial nerve VII). It also contains lymph channels. A posterior displacement of the condyle results in an increase in Tinnitus and Vertigo.
There is a mechanism that protects the inner ear form excessive sound. This protection is achieved by certain intratympanic muscles. Among them the tensor tympani muscle which is also supplied by a segment of the trigeminal nerve. Again when there is deep somatic and visceral pain occurring from a primary pain source such as the muscles of mastication, the effects spread to other divisions of the same neural segment. The action of tensor tympani is to pull on the tympanic membrane to increase its tension to reduce the movement of the ossicles when loud sound is present, hence protection of the middle ear. A dysfunction in this muscle will result in sensitivity to loud noise.




The EYE Connection

Patients suffering from craniomandibular dysfunction often complain of pain behind the eyes. The back portion of the orbital bony complex contains the greater and lesser wing of the sphenoid bone. The bones where the lateral and medial pterygoid muscles insert lie just below them namely medial and lateral pterygoid plates. Chronic contracture of these muscles could result in the torquing action of the sphenoids. Another recent finding that also could lead to retro-orbital pain is the Spenomandibular muscle. It originates from maxillar portion of the sphenoid bone and inserts into the internal oblique line of the mandible.



Monday, April 15, 2013

Bruxism In Children

Bruxism in Children

And recognizing the “Allergic Child”



A frequent question that I get asked by parents is “why my child is grinding”?
I am dedicating this to the late Dr. James F. Garry, a pioneering pediatric and TMJD dentist, the co-inventor of the “Nuk Sager Nipple”. And teacher and mentor to many dentists and doctors, including my self and my mentor Dr. Clayton Chan.

Studies have shown that 20% of non-allergic children were grinding teeth in comparison to 60% of children who were allergic. Dentists commonly believe that the cause of grinding is a “mind thing” due to local irritants, systemic factors, occupational factors, or a combination of these. Allergies as a factor have been ignored by many.
Listed are frequently seen facts that lead to bruxing:
  • Mouth breathing – causing dry mouth and lessened flow of saliva, thus diminishing the need for swallowing
  • Allergic itching or thickening of the palate and ears – bruxing relieves those symptoms
  • Negative pressure build up in the middle ear cavity with auditory tubule dysfunction causing swelling of the mucosa in the Eustachian tube
  • Eruption of secondary dentition and exfoliation of the primary and the attempt to establish contact of greater number of teeth

It is imperative to know that in a growing child that is hypersensitive like an allergic child, metabolic changes in the affected areas interfere with normal growth and development. The most common dental manifestation is malocclusion caused by chronic mouth breathing as a result of upper respiratory tract edema. This will lead to, if untreated, to a neuromuscular imbalance between the lower jaw and the upper jaw. The patient may accommodate for many years as he/she are growing, then suddenly develop myofacial pain dysfunction and TMJD.

Recognizing the allergic child: “Listen and Observe”!

  

Most allergic children can be recognized by the following:

  • Allergic shiners bellow the eyes (dark lines)
  • Intermittent hearing difficulties
  • Complaint of stuffy ears, full ears, popping ears, fullness of head, vertigo
  • Loss of smell and taste
  • Sounds of an allergic child: nasal speech, constantly clearing throat, wheezing, frequent gulping
  • Open mouth syndrome
  • Forward head posture
  • Shoulders are hanging and rolled forward
  • Body seems bend and collapsed
  • Adenoidal facial expression
  • Teeth of the upper jaw are forward and retrognathic lower jaw
  • Lower lips dry and rolled
  • Bed wetting
  • Snoring
  • Sleep apnea
  • Growth failure

In children, growth failure and short stature are the major manifestations of Growth Hormone deficiency, where the most intense period of growth hormone release is shortly after the onset of deep sleep. An allergic child that is a mouth breather has insufficient deep sleep stages.

As always, a primary diagnosis should come from your physician and, or pediatrician. A dentist’s role should aid in primary recognition and making the parent aware of these problems and recommend appropriate medical evaluation of the child. 

Saturday, April 13, 2013

IS SURGERY THE ONLY OPTION?

Over the years practicing orthodontics and treating mal-occlusions and TMJD (temporomandibular disorders), treatment of the Class III (under bite) type of mal-occlusion has been the most rewarding experience, having a patient or their parents thank me for saving them from surgery. My goal is to inform the consumer that there are alternatives to surgery, not always but in majority of cases.

What is a Class III mal-occlusion? 
Class III malocclusion is considered to be one of the most difficult and complex orthodontic problems to treat. Prevalence of class III malocclusion in Caucasians ranges from 0.8 to 4.0% and rises up to 1213% in Chinese and Japanese populations. Skeletal class III malocclusion may either be associated with a maxillary retrusion (shorter upper jaw), a mandibular protrusion (longer lower jaw), or a combination of the two.
A poor facial appearance is often the patient's chief complaint, but may be accompanied by functional problems, temporomandibular disorders, or psychosocial handicaps.




Majority of these cases end up as surgical cases. The treatment usually involves a bilateral saggital split osteotomy of the lower jaw and/or advancement of the upper jaw with a Le Fort I osteotomy with pre-surgical and post-surgical orthodontics.



The surgery usually takes about seven hours to perform. The post-surgical complications like any surgery are nausea, numb upper and lower jaw, swelling, bruising, drooling, nose bleeding. Patients are restricted to soft and blended food.

But is this the only treatment option?

Case Report:
Patient L.A. 11.5 years of age presented to my office in 2010 for a 3rd opinion on his orthodontic treatment. The two previous orthodontists had recommended to hold off with treatment until patient reaches 16 years of age and at that time commence with pre-surgical orthodontics and surgery once initial alignment was completed.
After gathering the necessary records I came to the conclusion that this case could be treated non-surgically. A final consultation was scheduled and I discussed my plan of action which included of:

Phase I, treatment with a modified Tandem appliance to promote growth of the maxillary jaw forward, which is indicated in the mixed dentition stage.



Phase II, commencement with straight wire braces and the addition of CS 2000 springs in later stages of treatment.


Patient's parents agreed to my recommendations and were happy that the possibility of surgery was reduced.
L.A. was scheduled for treatment and following are the before, during and after images. Treatment ended in Summer of 2012 and patient was recently seen for follow up and did not show any relapse.

Pre-treatment

Mid treatment 6 months treatment with
Tandem appliance


Phase 2 braces and CS 2000

Completion of treatment in 2012


Cephalometric comparisons from start to finish

Discussion:
This case report describes the non-surgical intervention and treatment of a skeletal Class III mal-occlusion. With the advancements in today's dental technology and treatment procedures, surgical intervention should not be the first resort for treatment. As this case shows normal skeletal base relationships and normal occlusion can be achieved in a less invasive procedure and non-surgically.
In conclusion, I am not advocating that surgery should not be an option, but it is imperative to seek multiple opinions before a final decision for treatment is decided upon.

The author:
My name is Ramin Mehregan D.M.D. I graduated in 1992 from Boston University School of Graduate Dentistry. In 1999 I started my training with Dr. Clayton Chan (www.occlusionconnections.com) in the field of Gneuromuscular Dentistry which focuses in the treatment of TMJD disorders and full mouth rehabilitation. Upon completion of my training I continued my training in the field of orthodontics/orthopedics. Gneuromuscular Occlusion/Dentistry combines the application of sound gnathologic occlusal mechanics of occlusion with scientific and physiologic principles of NMD (Neuro-muscular-dentistry) in a balanced manner. Gneuromuscular is a discipline of dentistry that is now being recognized as key to comprehensive TMD, restorative and orthodontic dentistry that has been the missing link in our dental society.